The research group leaders, the National Cancer Research Center of Madrid, Manuel · Serrano said, scientists have known that the antitumor protein P53 (also called Trp53) and it's an adjustment of ARF, involved in preventing and eliminating the process of cell injury. But new research has confirmed that both gene activity increases, also produced the "powerful" antioxidant effects, their functionality is not limited to fight cancer.
In the control of the mouse gene experiments found that both of these protein levels of growth not only destroys cancer tumor, you can postpone aging. Tests confirmed that using a large dose of the two genes in mice, more than any other mouse's longer life expectancy. Considering that cancer and aging both processes and cellular damage accumulation, which is understandable.
Cancer cell damage caused distortion exacerbate the cumulative results of gene, aging from gene damage aggravation and features missing (slow metabolism). Therefore, the added strong anticancer drugs, genes may also postpone aging.
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